The drug helps to release nitrogen oxide from the nerve endings located in the endothelium of cavernous bodies. Oxide, in turn, stimulates the synthesis of cyclic guanosine monophosphate in the cells of smooth muscle fibers markobader.blog.se/was-ist-erektile-dysfunktion/
. CGMF accumulates in the muscle tissue and causes its relaxation, increasing the blood flow to the carvaenous bodies. Thus, the process of inhibition of phosphodiesterase 5 is accompanied by an increase in erection and an increase in cGMP. The main condition for the above chain of reactions is sexual stimulation. In the absence of such activity, the active substance is ineffective.
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